Acute lung injury

Michelle Ng Gong; David C. Christiani

doi:10.1007/978-1-59259-963-9_25

Acute lung injury

Research output: Chapter in Book/Report/Conference proceeding › Chapter

Abstract

The acute respiratory distress syndrome (ARDS)/acute lung injury is a major cause of morbidity and mortality throughout the world. ARDS is an acute syndrome of lung inflammation and increased permeability associated with severe hypoxia and bilateral infiltrates on chest radiographs with no evidence of left heart failure. This chapter reviews the evidence for genetic determinants in acute lung injury and ARDS with a focus on potential candidate gene polymorphism that may be important in the development and outcome of ARDS.

Original language	English (US)
Title of host publication	Principles of Molecular Medicine
Publisher	Humana Press
Pages	231-238
Number of pages	8
ISBN (Print)	9781588292025
DOIs	https://doi.org/10.1007/978-1-59259-963-9_25
State	Published - 2006
Externally published	Yes

Keywords

Acute lung injury (ALI)
acute respiratory distress
angiotensin-converting enzyme (ACE)
glutathnione-S-transferase (GST)
syndrome (ARDS)

ASJC Scopus subject areas

General Medicine

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10.1007/978-1-59259-963-9_25

Cite this

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title = "Acute lung injury",

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AB - The acute respiratory distress syndrome (ARDS)/acute lung injury is a major cause of morbidity and mortality throughout the world. ARDS is an acute syndrome of lung inflammation and increased permeability associated with severe hypoxia and bilateral infiltrates on chest radiographs with no evidence of left heart failure. This chapter reviews the evidence for genetic determinants in acute lung injury and ARDS with a focus on potential candidate gene polymorphism that may be important in the development and outcome of ARDS.

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KW - acute respiratory distress

KW - angiotensin-converting enzyme (ACE)

KW - glutathnione-S-transferase (GST)

KW - syndrome (ARDS)

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BT - Principles of Molecular Medicine

PB - Humana Press

ER -

Acute lung injury

Abstract

Keywords

ASJC Scopus subject areas

Access to Document

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