Bernice E. Morrow, Ph.D.

Genetics

Phone718-678-1121
Emailbernice.morrow@einsteinmed.org
Websitehttp://www.einstein.yu.edu/faculty/4279/bernice-morrow/

Source: Scopus
Calculated based on no. of publications stored in Pure and citations from Scopus

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2 Active
9 Finished

Cell fate choices by Tbx1 in forming the mammalian heart
Morrow, B. E. & Zheng, D.
8/20/20 → 4/30/21
Project: Research project
Genetic Basis of Syndromic and Non-Syndromic Congential Heart Defects
Morrow, B. E., Emanuel, B. & Goldmuntz, E.
Eunice Kennedy Shriver National Institute of Child Health and Human Development
9/24/11 → 6/30/21
Project: Research project
Tbx1 and canonical Wnt signaling in the second heart field
Morrow, B. E. & Chenleng, C.
9/15/15 → 8/31/16
Project: Research project
Developmental Genetics of the Pharyngeal Apparatus
Morrow, B. E. & Rose, S. A.
7/1/09 → 6/30/12
Project: Research project
Sequence Variations in Low Copy Repeats on 22q11.2
Morrow, B. E. & Rose, S. A.
5/1/09 → 4/30/11
Project: Research project

10306 Citations
46 h-Index
132 Article
6 Review article
5 Comment/debate
2 Chapter
More
- 2 Letter

Complete Sequence of the 22q11.2 Allele in 1,053 Subjects with 22q11.2 Deletion Syndrome Reveals Modifiers of Conotruncal Heart Defects
International 22q11.2 Brain and Behavior Consortium, Jan 2 2020, In: American Journal of Human Genetics. 106, 1, p. 26-40 15 p.
Research output: Contribution to journal › Article › peer-review
10 Scopus citations
Gene-based analyses of the maternal genome implicate maternal effect genes as risk factors for conotruncal heart defects
Pediatric Cardiac Genomics Consortium, Jun 2020, In: PloS one. 15, 6, e0234357.
Research output: Contribution to journal › Article › peer-review

Open Access
1 Scopus citations
Genetic contributors to risk of schizophrenia in the presence of a 22q11.2 deletion
International 22q11.2DS Brain and Behavior Consortium, 2020, (Accepted/In press) In: Molecular Psychiatry.
Research output: Contribution to journal › Article › peer-review
19 Scopus citations
Optical mapping of the 22q11.2DS region reveals complex repeat structures and preferred locations for non-allelic homologous recombination (NAHR)
Pastor, S., Tran, O., Jin, A., Carrado, D., Silva, B. A., Uppuluri, L., Abid, H. Z., Young, E., Crowley, T. B., Bailey, A. G., McGinn, D. E., McDonald-McGinn, D. M., Zackai, E. H., Xie, M., Taylor, D., Morrow, B. E., Xiao, M. & Emanuel, B. S., Dec 1 2020, In: Scientific reports. 10, 1, 12235.
Research output: Contribution to journal › Article › peer-review

Open Access
3 Scopus citations
Using common genetic variation to examine phenotypic expression and risk prediction in 22q11.2 deletion syndrome
International 22q11.2 Brain and Behavior Consortium, Dec 2020, In: Nature Medicine. 26, 12, p. 1912-1918 7 p.
Research output: Contribution to journal › Article › peer-review
9 Scopus citations