The reparative function of cardiomyocytes in the infarcted myocardium

Nikolaos G. Frangogiannis

doi:10.1016/j.cmet.2015.05.015

The reparative function of cardiomyocytes in the infarcted myocardium

Nikolaos G. Frangogiannis

Medicine

Research output: Contribution to journal › Short survey › peer-review

9 Scopus citations

Abstract

Effective repair of the infarcted heart requires spatial containment of the inflammatory reaction within the infarcted area. A recently published study demonstrates that in the pro-inflammatory environment of the infarct border zone, surviving cardiomyocytes restrain inflammation by activating a Reg3β-dependent cascade that recruits reparative macrophages and promotes neutrophil clearance.

Original language	English (US)
Pages (from-to)	797-798
Number of pages	2
Journal	Cell metabolism
Volume	21
Issue number	6
DOIs	https://doi.org/10.1016/j.cmet.2015.05.015
State	Published - Jun 2 2015

ASJC Scopus subject areas

Physiology
Molecular Biology
Cell Biology

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10.1016/j.cmet.2015.05.015

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title = "The reparative function of cardiomyocytes in the infarcted myocardium",

abstract = "Effective repair of the infarcted heart requires spatial containment of the inflammatory reaction within the infarcted area. A recently published study demonstrates that in the pro-inflammatory environment of the infarct border zone, surviving cardiomyocytes restrain inflammation by activating a Reg3β-dependent cascade that recruits reparative macrophages and promotes neutrophil clearance.",

author = "Frangogiannis, {Nikolaos G.}",

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year = "2015",

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AU - Frangogiannis, Nikolaos G.

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N2 - Effective repair of the infarcted heart requires spatial containment of the inflammatory reaction within the infarcted area. A recently published study demonstrates that in the pro-inflammatory environment of the infarct border zone, surviving cardiomyocytes restrain inflammation by activating a Reg3β-dependent cascade that recruits reparative macrophages and promotes neutrophil clearance.

AB - Effective repair of the infarcted heart requires spatial containment of the inflammatory reaction within the infarcted area. A recently published study demonstrates that in the pro-inflammatory environment of the infarct border zone, surviving cardiomyocytes restrain inflammation by activating a Reg3β-dependent cascade that recruits reparative macrophages and promotes neutrophil clearance.

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DO - 10.1016/j.cmet.2015.05.015

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SP - 797

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JO - Cell metabolism

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The reparative function of cardiomyocytes in the infarcted myocardium

Abstract

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