Modulation of host cell metabolism by Trypanosoma cruzi

S. A. Morris; H. B. Tanowitz; J. P. Bilezikian; M. Wittner

doi:10.1016/0169-4758(91)90202-Y

Modulation of host cell metabolism by Trypanosoma cruzi

S. A. Morris, H. B. Tanowitz, J. P. Bilezikian, M. Wittner

Pathology

Research output: Contribution to journal › Article › peer-review

9 Scopus citations

Abstract

Chagos disease, caused by the hemoflagellate Trypanosoma cruzi, is a complicated and devastating disease. It is hypothesized that an important target of infection may be the endothelial cell and that both the acute and chronic forms of the disease involve abnormalities in the microcirculotion. Stephen Morris and colleagues suggest that endothelial cell dysfunction occurs as a consequence of amastigote-associated interference in host cell metabolism.

Original language	English (US)
Pages (from-to)	82-87
Number of pages	6
Journal	Parasitology Today
Volume	7
Issue number	4
DOIs	https://doi.org/10.1016/0169-4758(91)90202-Y
State	Published - 1991

ASJC Scopus subject areas

Parasitology

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10.1016/0169-4758(91)90202-Y

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title = "Modulation of host cell metabolism by Trypanosoma cruzi",

abstract = "Chagos disease, caused by the hemoflagellate Trypanosoma cruzi, is a complicated and devastating disease. It is hypothesized that an important target of infection may be the endothelial cell and that both the acute and chronic forms of the disease involve abnormalities in the microcirculotion. Stephen Morris and colleagues suggest that endothelial cell dysfunction occurs as a consequence of amastigote-associated interference in host cell metabolism.",

author = "Morris, {S. A.} and Tanowitz, {H. B.} and Bilezikian, {J. P.} and M. Wittner",

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AU - Morris, S. A.

AU - Tanowitz, H. B.

AU - Bilezikian, J. P.

AU - Wittner, M.

PY - 1991

Y1 - 1991

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AB - Chagos disease, caused by the hemoflagellate Trypanosoma cruzi, is a complicated and devastating disease. It is hypothesized that an important target of infection may be the endothelial cell and that both the acute and chronic forms of the disease involve abnormalities in the microcirculotion. Stephen Morris and colleagues suggest that endothelial cell dysfunction occurs as a consequence of amastigote-associated interference in host cell metabolism.

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Modulation of host cell metabolism by Trypanosoma cruzi

Abstract

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