Hypermagnesemia, Serum Calcium, and Parathyroid Hormone Levels

Thomas A. Wilson; Lalitha Gopalakrishnan; Gary Trager; Anoop Kapoor; I. N. Cholst; S. F. Steinberg; P. J. Tropper; H. E. Fox; G. V. Segre; J. P. Bilezikian

doi:10.1056/NEJM198408303110915

Hypermagnesemia, Serum Calcium, and Parathyroid Hormone Levels

Thomas A. Wilson, Lalitha Gopalakrishnan, Gary Trager, Anoop Kapoor, I. N. Cholst, S. F. Steinberg, P. J. Tropper, H. E. Fox, G. V. Segre, J. P. Bilezikian

Obstetrics & Gynecology and Women's Health

Research output: Contribution to journal › Letter

Abstract

To the Editor: Cholst et al. have shown that infusions of magnesium sulfate suppress serum calcium and parathyroid hormone concentrations in preeclamptic women (May 10 issue).¹ They suggest that the suppression of the serum calcium Concentration was due in part to suppression of parathyroid hormone secretion, but concede that “other factors may have contributed.” The evidence discussed below suggests to us that factors other than the suppression of parathyroid hormone were responsible for the hypocalcemia observed. The report indicates that the serum parathyroid hormone concentrations returned to base-line values at 180 minutes, while the serum calcium concentrations were at their.

Original language	English (US)
Number of pages	1
Journal	New England Journal of Medicine
Volume	311
Issue number	9
DOIs	https://doi.org/10.1056/NEJM198408303110915
State	Published - Aug 30 1984

ASJC Scopus subject areas

Medicine(all)

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10.1056/NEJM198408303110915

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Wilson, T. A., Gopalakrishnan, L., Trager, G., Kapoor, A., Cholst, I. N., Steinberg, S. F., Tropper, P. J., Fox, H. E., Segre, G. V., & Bilezikian, J. P. (1984). Hypermagnesemia, Serum Calcium, and Parathyroid Hormone Levels. New England Journal of Medicine, 311(9). https://doi.org/10.1056/NEJM198408303110915

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title = "Hypermagnesemia, Serum Calcium, and Parathyroid Hormone Levels",

abstract = "To the Editor: Cholst et al. have shown that infusions of magnesium sulfate suppress serum calcium and parathyroid hormone concentrations in preeclamptic women (May 10 issue).1 They suggest that the suppression of the serum calcium Concentration was due in part to suppression of parathyroid hormone secretion, but concede that “other factors may have contributed.” The evidence discussed below suggests to us that factors other than the suppression of parathyroid hormone were responsible for the hypocalcemia observed. The report indicates that the serum parathyroid hormone concentrations returned to base-line values at 180 minutes, while the serum calcium concentrations were at their.",

author = "Wilson, {Thomas A.} and Lalitha Gopalakrishnan and Gary Trager and Anoop Kapoor and Cholst, {I. N.} and Steinberg, {S. F.} and Tropper, {P. J.} and Fox, {H. E.} and Segre, {G. V.} and Bilezikian, {J. P.}",

year = "1984",

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T1 - Hypermagnesemia, Serum Calcium, and Parathyroid Hormone Levels

AU - Wilson, Thomas A.

AU - Gopalakrishnan, Lalitha

AU - Trager, Gary

AU - Kapoor, Anoop

AU - Cholst, I. N.

AU - Steinberg, S. F.

AU - Tropper, P. J.

AU - Fox, H. E.

AU - Segre, G. V.

AU - Bilezikian, J. P.

PY - 1984/8/30

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N2 - To the Editor: Cholst et al. have shown that infusions of magnesium sulfate suppress serum calcium and parathyroid hormone concentrations in preeclamptic women (May 10 issue).1 They suggest that the suppression of the serum calcium Concentration was due in part to suppression of parathyroid hormone secretion, but concede that “other factors may have contributed.” The evidence discussed below suggests to us that factors other than the suppression of parathyroid hormone were responsible for the hypocalcemia observed. The report indicates that the serum parathyroid hormone concentrations returned to base-line values at 180 minutes, while the serum calcium concentrations were at their.

AB - To the Editor: Cholst et al. have shown that infusions of magnesium sulfate suppress serum calcium and parathyroid hormone concentrations in preeclamptic women (May 10 issue).1 They suggest that the suppression of the serum calcium Concentration was due in part to suppression of parathyroid hormone secretion, but concede that “other factors may have contributed.” The evidence discussed below suggests to us that factors other than the suppression of parathyroid hormone were responsible for the hypocalcemia observed. The report indicates that the serum parathyroid hormone concentrations returned to base-line values at 180 minutes, while the serum calcium concentrations were at their.

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