Green Tea (Camellia sinensis) ameliorates retinopathy and renal mitochondrial defects but deteriorates collagen glycoxidation and cross-linking in experimental diabetes

Georgian T. Mustata, Mariana G. Rosca, Oliver Reihl, Klaus Biemel, Mark A. Smith, Ashwini Viswanathan, Christopher Strauch, Yun Peng Du, Timothy S. Kern, Markus Lederer, Michael Brownlee, Miriam F. Weiss, Vincent M. Monnier

Research output: Contribution to journalArticle

Original languageEnglish (US)
Number of pages1
JournalAnnals of the New York Academy of Sciences
Volume1043
Issue number1
DOIs
StatePublished - Jan 1 2005

ASJC Scopus subject areas

  • Neuroscience(all)
  • Biochemistry, Genetics and Molecular Biology(all)
  • History and Philosophy of Science

Cite this

Mustata, G. T., Rosca, M. G., Reihl, O., Biemel, K., Smith, M. A., Viswanathan, A., Strauch, C., Du, Y. P., Kern, T. S., Lederer, M., Brownlee, M., Weiss, M. F., & Monnier, V. M. (2005). Green Tea (Camellia sinensis) ameliorates retinopathy and renal mitochondrial defects but deteriorates collagen glycoxidation and cross-linking in experimental diabetes. Annals of the New York Academy of Sciences, 1043(1). https://doi.org/10.1196/annals.1333.154