Dissecting risk haplotypes in sporadic Alzheimer's disease

Frank Soldner, Rudolf Jaenisch

Research output: Contribution to journalShort surveypeer-review

6 Scopus citations


Understanding how genetic risk variants contribute to complex diseases is crucial for predicting disease susceptibility and developing patient-tailored therapies. In this issue of Cell Stem Cell, Young et al. (2015) dissect the function of common non-coding risk haplotypes in the SORL1 locus in the pathogenesis of sporadic Alzheimer's disease using patient-derived induced pluripotent stem cells.

Original languageEnglish (US)
Pages (from-to)341-342
Number of pages2
JournalCell Stem Cell
Issue number4
StatePublished - Apr 2 2015

ASJC Scopus subject areas

  • Molecular Medicine
  • Genetics
  • Cell Biology

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