Skip to main navigation
Skip to search
Skip to main content
Albert Einstein College of Medicine Home
Home
Faculty
Departments
Grants
Publications
Prizes
Search by expertise, name or affiliation
Defects in G protein-coupled signal transduction in human disease
A. M. Spiegel
Research output
:
Contribution to journal
›
Review article
›
peer-review
188
Scopus citations
Overview
Fingerprint
Fingerprint
Dive into the research topics of 'Defects in G protein-coupled signal transduction in human disease'. Together they form a unique fingerprint.
Sort by
Weight
Alphabetically
Medicine & Life Sciences
GTP-Binding Proteins
100%
Signal Transduction
78%
Guanosine Triphosphate
26%
G-Protein-Coupled Receptors
25%
Familial Glucocorticoid Deficiency 1
24%
Hypocalciuric hypercalcemia, familial, type 1
20%
Cone Opsins
20%
Corticotropin Receptors
20%
Nephrogenic Diabetes Insipidus
18%
Color Vision Defects
18%
Bacterial Toxins
17%
LH Receptors
16%
Thyrotropin Receptors
16%
Calcium-Sensing Receptors
16%
Vasopressin Receptors
16%
Rhodopsin
15%
Cholera
15%
Guanine Nucleotides
15%
Retinitis Pigmentosa
15%
Whooping Cough
15%
GTP Phosphohydrolases
14%
Loss of Function Mutation
14%
Second Messenger Systems
13%
Missense Mutation
12%
Neurotransmitter Agents
10%
Hormones
8%
Peptides
7%
Membranes
7%
Mutation
7%