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Defects in G protein-coupled signal transduction in human disease
A. M. Spiegel
Research output
:
Contribution to journal
›
Review article
›
peer-review
187
Scopus citations
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Dive into the research topics of 'Defects in G protein-coupled signal transduction in human disease'. Together they form a unique fingerprint.
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Medicine & Life Sciences
GTP-Binding Proteins
100%
Signal Transduction
78%
Guanosine Triphosphate
26%
G-Protein-Coupled Receptors
25%
Familial Glucocorticoid Deficiency 1
24%
Hypocalciuric hypercalcemia, familial, type 1
20%
Cone Opsins
20%
Corticotropin Receptors
20%
Nephrogenic Diabetes Insipidus
18%
Color Vision Defects
18%
Bacterial Toxins
17%
LH Receptors
16%
Thyrotropin Receptors
16%
Calcium-Sensing Receptors
16%
Vasopressin Receptors
16%
Rhodopsin
15%
Cholera
15%
Guanine Nucleotides
15%
Retinitis Pigmentosa
15%
Whooping Cough
15%
GTP Phosphohydrolases
14%
Loss of Function Mutation
14%
Second Messenger Systems
13%
Missense Mutation
12%
Neurotransmitter Agents
10%
Hormones
8%
Peptides
7%
Membranes
7%
Mutation
7%