Defective annexin A5 crystallization: A mechanism for pregnancy losses in the antiphospholipid syndrome

J. Rand, P. V. Eerden, X. X. Wu, C. Chazotte

Research output: Contribution to journalArticle

10 Scopus citations

Abstract

We have accumulated a significant body of evidence that support the hypothesis that aPL antibody-mediated disruption of annexin A5 crystallization is a mechanism for RSPL in the APS. Further investigation of the functions of the annexins in placental physiology and disease are likely to be productive areas for future research.

Original languageEnglish (US)
Pages (from-to)77-81
Number of pages5
JournalThrombosis Research
Volume115
Issue numberSUPPL.
Publication statusPublished - Feb 1 2005

    Fingerprint

ASJC Scopus subject areas

  • Hematology

Cite this

Rand, J., Eerden, P. V., Wu, X. X., & Chazotte, C. (2005). Defective annexin A5 crystallization: A mechanism for pregnancy losses in the antiphospholipid syndrome. Thrombosis Research, 115(SUPPL.), 77-81.