We have accumulated a significant body of evidence that support the hypothesis that aPL antibody-mediated disruption of annexin A5 crystallization is a mechanism for RSPL in the APS. Further investigation of the functions of the annexins in placental physiology and disease are likely to be productive areas for future research.
|Original language||English (US)|
|Number of pages||5|
|State||Published - Feb 2005|
ASJC Scopus subject areas